Biobank Dataset by Pathology

This document has been developed within the framework of COST Action CA20121 (BenBedPhar), which aims to foster the transition from basic knowledge on the transcription factor NRF2 towards pharmacological and clinical applications in noncommunicable diseases (NCDs). Through its Working Groups1 and 3, BenBedPhar promotes the harmonization of biological sample collections, the standardization of procedures, and the establishment of technological platforms that facilitate access to human materials and metadata relevant to NRF2-related research. The integrated biobank dataset presented here represents a tangible outcome of these coordinated efforts. It compiles and harmonizes information from multiple European biobanks and research cohorts, organized by pathology, providing a unified view of available biospecimens, cohort structures, and demographic
variables. By linking biobank resources to BenBedPhar’s objectives in pharmacological regulation of NRF2, this deliverable creates a foundation for standardized access to biospecimens and data across Europe and strengthens the network’s capacity for translational research.
This integration supports the Action’s broader strategy to connect academic, clinical, and industrial sectors through harmonized infrastructures, promoting cross-disease analyses, biomarker validation, and therapeutic development. Continued updates of this dataset will further enhance interoperability and sustainability within the BenBedPhar network, facilitating future preclinical and
clinical research on NRF2-related mechanisms in major NCDs.

Cohort owner (Cohort name)Gothenburg H70 Birth Cohort Studies (Anna Zettergren)
Type of recruitmentPopulation-based longitudinal study of older individuals, recruited from the general population based on birth year and date.
Place of recruitmentGothenburg, Sweden
Number of subjectsApprox. 10,000 (but much of the data is only available in sub-groups)
F/MApprox 60% females
Age range70-110
Healthy/disease (stage?)Population-based cohorts (baseline age varies between 70 and 95 years of age)
Medications y/nSelf-reported information on medication
Lifestyle
Dietary intakeDiet history interview (data on foods, energy and nutrient intake)
Physical activityyes; interview and self-rating questionnaire (there are also measures of physical function, such as grip strength, gait speed, balance.
Biomarkers
Body compositionDXA, Antropometric measures
InflammationCRP (not high sensitive)
Vascular/adipose tissue
CV healthNT-proBNP
KidneyCreatinine
LiverAST, ALT
Lipid metabolismTotal-, HDL-, LDL-Cholesterol, Triglycerides
Glucose metabolism
Protein statusN/A
Metabolomics
Iron status Hemoglobin
Vitamin & trace element statusHomocysteine (indirect measure of vit B12)
Fatty Acids
AGEsN/A
Glycation
ROS/redox status
Lipidperoxidation
Others (please add)Genetic data (i.e. GWAS), CSF-biomarkers for Alzheimer´s disease and neurodegeneration (Abeta42, t-tau, p-tau, NfL, Neurogranin), plasma-NfL, Neuroimaging
Samples available for further use
Plasma/serumyes
Spot urineN/A
24h urineN/A
Pax tubes (or isolated RNA)N/A
Cells N/A
Tissues N/A
others
GeneticsGWAS
Transcription Factors
NRF2/KEAP1 status
redox gene status
CSF Biomarkers
NeuroimagingAbeta42, t-tau, p-tau, NfL, Neurogranin

Within the scope of BenBedPhar, the Ageing datasets contribute to understanding how NRF2-related mechanisms intersect with disease-specific pathways and clinical phenotypes. This integrated resource compiles approximately 51 entries from 0 biobank sources, representing collections from 0 countries. Sample matrices include a wide range of biological materials, which
collectively support translational analyses, biomarker discovery, and cross-disease comparison. By aligning this information under a harmonized structure, the Action strengthens its technological foundation for data-driven pharmacological innovation and facilitates coordination among research groups involved in preclinical and clinical NRF2 studies.

Alzheimer disease

Cohort owner (Cohort name)Spanish-Romanian cohort (Gina Manda)
Type of recruitmentCase-control: 40 AD (T0 baseline) and 40 controls
Longitudinal: 40 AD (T0) and 32 (T1 after 1 years)
Place of recruitmentSpain (Hospital Universitari Santa Maria-IRB Leida, Lleida)
Number of subjects80
F/M28F/52M
Age range45-86
Healthy/disease (stage?)40 Alzheimer/40 controls
Medications y/nAD treatment available
Lifestyle
Dietary intakeN/A
Physical activityN/A
Biomarkers
Body composition
InflammationWhole blood Gene expression of 168 NFKB-related genes and 84 REDOX genes for 80 individuals (Array 1, Array 2, Array 3)
Vascular/adipose tissue
CV healthN/A
KidneyN/A
LiverN/A
Lipid metabolismN/A
Glucose metabolism
Protein statusN/A
Metabolomics
Iron status N/A
Vitamin & trace element statusN/A
Fatty Acids
AGEsN/A
Glycation
ROS/redox status
Lipidperoxidation
Others (please add)“For AD patients APOE genotype; CSF biomarkers TAU, pTAU, Aβ(1–40), plasma Total TAU, Aβ(1–40) and
Aβ(1–42) https//www.dovepress.com/getfile.php?fileID=76016″
Samples available for further use
Plasma/serumN/A
Spot urineN/A
24h urineN/A
Pax tubes (or isolated RNA)112
Cells N/A
Tissues N/A
others
Genetics
Transcription Factors
NRF2/KEAP1 status
redox gene status
CSF Biomarkers
Neuroimaging
Cohort owner (Cohort name)Alzheimer’s disease Cohort (Anna Zettergren)
Type of recruitmentClinical sample, baseline only (not followed over time)
Place of recruitmentSwedish Memory Clinics
Number of subjectsApprox. 500
F/MApprox 60% females
Age rangemean age at onset 75 years
Healthy/disease (stage?)Alzheimer’s Disease
Medications y/nN/A
Lifestyle
Dietary intakeN/A
Physical activityN/A
Biomarkers
Body compositionN/A
InflammationN/A
Vascular/adipose tissue
CV healthN/A
KidneyN/A
LiverN/A
Lipid metabolismN/A
Glucose metabolism
Protein statusN/A
Metabolomics
Iron status N/A
Vitamin & trace element statusN/A
Fatty Acids
AGEsN/A
Glycation
ROS/redox status
Lipidperoxidation
Others (please add)Genetic data (i.e. GWAS), CSF-biomarkers for Alzheimer´s disease and neurodegeneration (Abeta42, t-tau, p-tau)
Samples available for further use
Plasma/serumN/A
Spot urineN/A
24h urineN/A
Pax tubes (or isolated RNA)N/A
Cells N/A
Tissues N/A
others
Genetics
Transcription Factors
NRF2/KEAP1 status
redox gene status
CSF Biomarkers
Neuroimaging

Within the scope of BenBedPhar, the Alzheimer disease datasets contribute to understanding how NRF2-related mechanisms intersect with disease-specific pathways and clinical phenotypes. This integrated resource compiles approximately 102 entries from 0 biobank sources, representing collections from 0 countries. Sample matrices include a wide range of biological materials, which collectively support translational analyses, biomarker discovery, and cross-disease comparison. By aligning this information under a harmonized structure, the Action strengthens its technological foundation for data-driven pharmacological innovation and facilitates coordination among research groups involved in preclinical and clinical NRF2 studies.

Cardiovascular disease

Cohort owner (Cohort name)Cardio_Adipose tissues (Eugenia Carvalho)
Type of recruitment
Place of recruitmentCardiothroracic Surgery Unit at the University Hospital of Coimbra, Coimbra, Portugal
Number of subjects36 (14 with diabetes)
F/M33/3
Age range55-85
Healthy/disease (stage?)Patientes elected for cardiac disease, with or withouth diabetes
Medications y/nyes (antiplatelets, antirihmics, anticoagulants, insulin, alpha glucosidase inhibitors, DPP4 inhibitors, metformin, beta blcokers, calcium channel blocker, angiotesnin receptor blockers, angiotensin inhibitors, diurectic, statins, vasodilectors)
Lifestyle
Dietary intakesmoking and drinking habits
Physical activityno
Biomarkers
Body compositionyes: height, weight and BMI
Inflammation
Vascular/adipose tissue
CV healthINR (internantional normalized ratio) and CK (creatine Kinase)
KidneyUrea; Creatinine
LiverAST (aspartate aminotrasferase);ALT (alinino transferase);GGT; Alkaline Phosphatase (ALP); LDH (lactate dehidrogenase)
Lipid metabolismDiagnose of dyslipidemia
Glucose metabolismFasting glucose levels
Protein status
Metabolomics
Iron status
Vitamin & trace element status
Fatty Acids
AGEs
Glycation
ROS/redox status
Lipidperoxidation
Others (please add)Blood pressure and diagnose of hipertension; type of cardioascular disease (coronary artery disease or valvule dysfunction or heart congenit disease)
Samples available for further use
Plasma/serum
Spot urine
24h urine
Pax tubes (or isolated RNA)
Cells
Tissues X (epicardial and subcutaneous adipose tissue)
others
Genetics
Transcription Factors
NRF2/KEAP1 status
redox gene status
CSF Biomarkers
Neuroimaging
Cohort owner (Cohort name)Cardio_Biofluids ( Eugenia Carvalho)
Type of recruitment
Place of recruitmentCardiothroracic Surgery Unit at the University Hospital of Coimbra, Coimbra, Portugal
Number of subjects191 (75 with diabetes)
F/M51/140
Age range55-85
Healthy/disease (stage?)Patientes elected for cardiac disease, with or withouth diabetes
Medications y/nyes (antiplatelets, antirihmics, anticoagulants, insulin, alpha glucosidase inhibitors, DPP4 inhibitors, metformin, beta blockers, calcium channel blockers, angiotesnin receptor blockers, angiotensin inhibitors, diurectic, statins, vasodilectors)
Lifestyle
Dietary intakesmoking and drinking habits
Physical activityno
Biomarkers
Body compositionyes: height, weight and BMI
Inflammation
Vascular/adipose tissue
CV healthINR (internantional normalized ratio) and CK (creatine Kinase)
KidneyUrea; Creatinine
LiverAST (aspartate aminotrasferase);ALT (alinino transferase);GGT; Alkaline Phosphatase (ALP); LDH (lactate dehidrogenase)
Lipid metabolismDiagnose of dyslipidemia
Glucose metabolismFasting glucose levels
Protein status
Metabolomics
Iron status
Vitamin & trace element status
Fatty Acids
AGEs
Glycation
ROS/redox status
Lipidperoxidation
Others (please add)Blood pressure and diagnose of hipertension; type of cardioascular disease (coronary artery disease or valvule dysfunction or heart congenit disease)
Samples available for further use
Plasma/serumX (and whole blood)
Spot urine
24h urine
Pax tubes (or isolated RNA)
Cells
Tissues
others
Genetics
Transcription Factors
NRF2/KEAP1 status
redox gene status
CSF Biomarkers
Neuroimaging
Cohort owner (Cohort name)Cardio_Internal thoracic artery (Eugenia Carvalho)
Type of recruitment
Place of recruitmentCardiothroracic Surgery Unit at the University Hospital of Coimbra, Coimbra, Portugal
Number of subjects31 (14 with diabetes)
F/M29/2
Age range55-85
Healthy/disease (stage?)Patientes elected for cardiac disease, with or withouth diabetes
Medications y/nyes (antiplatelets, antirihmics, anticoagulants, insulin, alpha glucosidase inhibitors, DPP4 inhibitors, metformin, beta blcokers, calcium channel blocker, angiotesnin receptor blockers, angiotensin inhibitors, diurectic, statins, vasodilectors)
Lifestyle
Dietary intakesmoking and drinking habits
Physical activityno
Biomarkers
Body compositionyes: height, weight and BMI
Inflammation
Vascular/adipose tissue
CV healthINR (internantional normalized ratio) and CK (creatine Kinase)
KidneyUrea; Creatinine
LiverAST (aspartate aminotrasferase);ALT (alinino transferase);GGT; Alkaline Phosphatase (ALP); LDH (lactate dehidrogenase)
Lipid metabolismDiagnose of dyslipidemia
Glucose metabolismFasting glucose levels
Protein status
Metabolomics
Iron status
Vitamin & trace element status
Fatty Acids
AGEs
Glycation
ROS/redox status
Lipidperoxidation
Others (please add)Blood pressure and diagnose of hipertension; type of cardioascular disease (coronary artery disease or valvule dysfunction or heart congenit disease)
Samples available for further use
Plasma/serumX
Spot urine
24h urine
Pax tubes (or isolated RNA)
Cells
Tissues X (internal thoracic artery)
others
Genetics
Transcription Factors
NRF2/KEAP1 status
redox gene status
CSF Biomarkers
Neuroimaging

Within the scope of BenBedPhar, the Cardiovascular disease datasets contribute to understanding how NRF2-related mechanisms intersect with disease-specific pathways and clinical phenotypes. This integrated resource compiles approximately 153 entries from 0 biobank sources, representing collections from 0 countries. Sample matrices include a wide range of biological materials, which collectively support translational analyses, biomarker discovery, and cross-disease comparison. By aligning this information under a harmonized structure, the Action strengthens its technological foundation for data-driven pharmacological innovation and facilitates coordination among research groups involved in preclinical and clinical NRF2 studies.

Cognitive impairment

Cohort owner (Cohort name)Romania (Gina Manda)
Type of recruitmentLongitudinal: 150 baseline (T0); 61 after 1month (T1); 32 after 6 months (T2)
Place of recruitmentRomania (National Institute of Pathology Victor Babes)
Number of subjects150
F/M101F/49M
Age range28-88
Healthy/disease (stage?)50 cognitive impairment/100 normal cognition
Medications y/nInfo available in Database
Lifestyle
Dietary intakeN/A
Physical activityN/A
Biomarkers
Body compositionOnly BMI is available for 112 individuals
InflammationWhole blood Gene expression of 84 NFKB-related genes and 84 REDOX genes for 76 individuals (Array 1, Array 3)
Vascular/adipose tissue
CV healthCytokines/chemokines/growth factors and CVD markers assessed in serum from 46 individuals (https//www.dovepress.com/getfile.php?fileID=66782)
KidneyN/A
LiverN/A
Lipid metabolismN/A
Glucose metabolism
Protein statusN/A
Metabolomics
Iron status N/A
Vitamin & trace element statusN/A
Fatty Acids
AGEsN/A
Glycation
ROS/redox status
Lipidperoxidation
Others (please add)
Samples available for further use
Plasma/serumAll available
Spot urineN/A
24h urineN/A
Pax tubes (or isolated RNA)All available
Cells PBMC or lymphocytes
Tissues N/A
others
Genetics
Transcription Factors
NRF2/KEAP1 status
redox gene status
CSF Biomarkers
Neuroimaging

Within the scope of BenBedPhar, the Cognitive impairment datasets contribute to understanding how NRF2-related mechanisms intersect with disease-specific pathways and clinical phenotypes. This integrated resource compiles approximately 51 entries from 0 biobank sources, representing collections from 0 countries. Sample matrices include a wide range of biological materials, which collectively support translational analyses, biomarker discovery, and cross-disease comparison. By aligning this information under a harmonized structure, the Action strengthens its technological foundation for data-driven pharmacological innovation and facilitates coordination among research groups involved in preclinical and clinical NRF2 studies.

Hepatocellular carcinoma

Cohort owner (Cohort name)Jordi Muntané
Type of recruitmentHepatocellular carcinoma
Place of recruitmentInstitute of Biomedicine of Seville (Spain)
Number of subjects409
F/M20/80
Age range13-90
Healthy/disease (stage?)Stages A-D
Medications y/nTransplantation, resection, radiofrequency, ablation, chemotherapy and systemic therapy
Lifestyle
Dietary intakeClinical history
Physical activityClinical history
Biomarkers
Body compositionClinical history
InflammationClinical history
Vascular/adipose tissueClinical history
CV healthClinical history
KidneyClinical history
LiverEtiology, cirrhosis
Lipid metabolismClinical history
Glucose metabolismClinical history
Protein statusClinical history
MetabolomicsClinical history
Iron status Clinical history
Vitamin & trace element statusClinical history
Fatty AcidsClinical history
AGEsNo available
GlycationNo available
ROS/redox statusNo available
LipidperoxidationNo available
Others (please add)
Samples available for further use
Plasma/serumYes
Spot urineNo
24h urineNo
Pax tubes (or isolated RNA)miRNA
Cells Buffy coat/PBMCs (storage -80°C), Whole blood/Packed cells
Tissues Cryopreserved tissue, Formalin/OCT-fixed Paraffin embedded tissue
others
GeneticsNo available
Transcription FactorsNo available
NRF2/KEAP1 statusNo available
redox gene statusNo available
CSF BiomarkersNo available
NeuroimagingClinical history

Within the scope of BenBedPhar, the Hepatocellular carcinoma datasets contribute to understanding how NRF2-related mechanisms intersect with disease-specific pathways and clinical phenotypes. This integrated resource compiles approximately 51 entries from 0 biobank sources, representing collections from 0 countries. Sample matrices include a wide range of biological materials, which collectively support translational analyses, biomarker discovery, and cross-disease comparison. By aligning this information under a harmonized structure, the Action strengthens its technological foundation for data-driven pharmacological innovation and facilitates coordination among research groups involved in preclinical and clinical NRF2 studies.

Obesity & Metabolic/Bariatric Surgery

Cohort owner (Cohort name)CHUC (Eugenia Carvalho)
Type of recruitmentObesity Consultation
Place of recruitmentCHUC
Number of subjects92 (18 with diabetes)
F/M78/14
Age range24-66
Healthy/disease (stage?)Healthy or type 2 diabetes; all obese
Medications y/nyes (metformin)
Lifestyle
Dietary intakenot evaluated
Physical activitynot evaluated
Biomarkers
Body compositionyes: height, weight and BMI
Inflammationyes: C-reactive protein
Vascular/adipose tissueyes: leptin, adiponectin
CV healthno
Kidneyno
Liveryes: AST (aspartate aminotrasferase)
ALT (alinino transferase)
Lipid metabolismyes: Triglycerides; LDL; HDL; Total Cholesterol
Glucose metabolismyes: Fasting Glucose; Fasting insulin; fasting c-peptide; HbA1C
Protein statusno
Metabolomicsno
Iron statusno
Vitamin & trace element statusno
Fatty Acidsyes
AGEsno
Glycationyes: plasma levels of GLO-1
ROS/redox statusno
Lipidperoxidationno
Others (please add) 
Samples available for further use
Plasma/serumyes: plasma/serum
Spot urineno
24h urineno
Pax tubes (or isolated RNA)yes: isolated RNA of Visceral adipose tissue
Cellsno
Tissuesyes: Subcutaneous and Visceral Adipose tissue
others 
Geneticsno
Transcription Factorsyes
NRF2/KEAP1 statusyes (NRF2 mRNA in Visceral adipose tissue)
redox gene statusyes (catalase, gss, gsr expression in Visceral adipose tissue)
CSF Biomarkersno
Neuroimagingno
Cohort owner (Cohort name)Metabolic Surgery (Eugenia Carvalho)
Type of recruitment 
Place of recruitmentGeneral Surgery Department at CHUC
Number of subjects57 ( 23 with diabetes)
F/M38/19
Age range18-65
Healthy/disease (stage?)People with Obesity
Medications y/nyes
Lifestyle
Dietary intakeNo information
Physical activityNo information
Biomarkers
Body compositionyes: height, weight and BMI, waist circunference, hip circunference and neck circunference
Inflammationyes: C-reactive protein
Vascular/adipose tissueNo
CV healthNo
Kidneyyes: Creatinine
Liveryes: AST (aspartate aminotrasferase)
ALT (alinino transferase)
LDH (lactate dehidrogenase)
Lipid metabolismTriglycerides; LDL; HDL; Total Cholesterol
Glucose metabolismFasting Glucose; Fasting insulin; fasting c-peptide; HbA1C
Protein statusno
Metabolomicsno
Iron statusno
Vitamin & trace element statusno
Fatty Acidsno
AGEsno
Glycationno
ROS/redox statusno
Lipidperoxidationno
Others (please add)Blood pressure
Samples available for further use
Plasma/serumplasma/serum/whole blood
Spot urineno
24h urineno
Pax tubes (or isolated RNA)no
Cellsno
TissuesSubcutaneous and Visceral Adipose tissue
othersno
Geneticsno
Transcription Factorsno
NRF2/KEAP1 statusno
redox gene statusno
CSF Biomarkersno
Neuroimagingno
Cohort owner (Cohort name)Metabolic Surgery (Eugenia Carvalho)
Type of recruitment 
Place of recruitmentGeneral Surgery Department at CHUC
Number of subjects57 ( 23 with diabetes)
F/M38/19
Age range18-65
Healthy/disease (stage?)People with Obesity
Medications y/nyes
Lifestyle
Dietary intakeNo information
Physical activityNo information
Biomarkers
Body compositionyes: height, weight and BMI, waist circunference, hip circunference and neck circunference
Inflammationyes: C-reactive protein
Vascular/adipose tissueNo
CV healthNo
Kidneyyes: Creatinine
Liveryes: AST (aspartate aminotrasferase)
ALT (alinino transferase)
LDH (lactate dehidrogenase)
Lipid metabolismTriglycerides; LDL; HDL; Total Cholesterol
Glucose metabolismFasting Glucose; Fasting insulin; fasting c-peptide; HbA1C
Protein statusno
Metabolomicsno
Iron statusno
Vitamin & trace element statusno
Fatty Acidsno
AGEsno
Glycationno
ROS/redox statusno
Lipidperoxidationno
Others (please add)Blood pressure
Samples available for further use
Plasma/serumplasma/serum/whole blood
Spot urineno
24h urineno
Pax tubes (or isolated RNA)no
Cellsno
TissuesSubcutaneous and Visceral Adipose tissue
othersno
Geneticsno
Transcription Factorsno
NRF2/KEAP1 statusno
redox gene statusno
CSF Biomarkersno
Neuroimagingno

Within the scope of BenBedPhar, the Pulmonary hypertension datasets contribute to understanding how NRF2-related mechanisms intersect with disease-specific pathways and clinical phenotypes. This integrated resource compiles approximately 51 entries from 0 biobank sources, representing collections from 0 countries. Sample matrices include a wide range of biological materials, which collectively support translational analyses, biomarker discovery, and cross-disease comparison. By aligning this information under a harmonized structure, the Action strengthens its technological foundation for data-driven pharmacological innovation and facilitates coordination among research groups involved in preclinical and clinical NRF2 studies.

Pulmonary hypertension

Cohort owner (Cohort name)María José Sánchez-Quintero
Type of recruitment
Place of recruitmentHospital Virgen de la Victoria, Málaga, Spain. Hospital 12 octubre, Madrid, Spain.
Number of subjects15-20
F/MBoth
Age range
Healthy/disease (stage?)Pulmonary Hypertension
Medications y/n
Lifestyle
Dietary intake
Physical activity
Biomarkers
Body composition
Inflammation
Vascular/adipose tissue
CV health
Kidney
Liver
Lipid metabolism
Glucose metabolism
Protein status
Metabolomics
Iron status
Vitamin & trace element status
Fatty Acids
AGEs
Glycation
ROS/redox status
Lipidperoxidation
Others (please add)
Samples available for further use
Plasma/serumyes
Spot urine
24h urine
Pax tubes (or isolated RNA)
Cells yes. Fibroblasts, PBMCs.
Tissues
others
Genetics
Transcription Factors
NRF2/KEAP1 status
redox gene status
CSF Biomarkers
Neuroimaging

Within the scope of BenBedPhar, the Pulmonary hypertension datasets contribute to understanding how NRF2-related mechanisms intersect with disease-specific pathways and clinical phenotypes. This integrated resource compiles approximately 51 entries from 0 biobank sources, representing collections from 0 countries. Sample matrices include a wide range of biological materials, which collectively support translational analyses, biomarker discovery, and cross-disease comparison. By aligning this information under a harmonized structure, the Action strengthens its technological foundation for data-driven pharmacological innovation and facilitates coordination among research groups involved in preclinical and clinical NRF2 studies.

Various diseases

Cohort owner (Cohort name)Ioannis Trougakos
Type of recruitmentIn the clinic, public media
Place of recruitmentAthens, Greece
Number of subjects>2500
F/M~50% M, 50% F
Age range18-92
Healthy/disease (stage?)Health and various diseases
Medications y/nDisease based / not in healthy
Lifestyle
Dietary intakeVarious (based also on health status)
Physical activityVarious (based also on health status)
Biomarkers
Body compositionVarious
InflammationN/A
Vascular/adipose tissueN/A
CV healthN/A
KidneyN/A
LiverN/A
Lipid metabolismN/A
Glucose metabolismN/A
Protein statusN/A
MetabolomicsN/A
Iron status N/A
Vitamin & trace element statusN/A
Fatty AcidsN/A
AGEsN/A
GlycationN/A
ROS/redox statusN/A
LipidperoxidationN/A
Others (please add)N/A
Samples available for further use
Plasma/serumYes
Spot urine
24h urine
Pax tubes (or isolated RNA)Isolated RNA
Cells
Tissues
others
GeneticsNot known
Transcription FactorsNot known
NRF2/KEAP1 statusNot known
redox gene statusNot known
CSF BiomarkersNot known
NeuroimagingNot known

Within the scope of BenBedPhar, the Various diseases datasets contribute to understanding how NRF2-related mechanisms intersect with disease-specific pathways and clinical phenotypes. This integrated resource compiles approximately 51 entries from 0 biobank sources, representing collections from 0 countries. Sample matrices include a wide range of biological materials, which collectively support translational analyses, biomarker discovery, and cross-disease comparison. By aligning this information under a harmonized structure, the Action strengthens its technological foundation for data-driven pharmacological innovation and facilitates coordination among research groups involved in preclinical and clinical NRF2 studies.